Neuroinflammation In Alzheimer's Disease Pathway

Alzheimer's disease is traditionally identified by the presence of misfolded aggregates of amyloid-beta (Aß) and tau proteins, which form plaques and tangles within the brain. These abnormal protein structures disrupt neural communication and lead to the progressive cognitive decline associated with the disease. However, recent research has highlighted the significant role of the body's innate immune system and neuroinflammation in the development and progression of Alzheimer's. This emerging evidence suggests that targeting neuroinflammation and modulating the immune response could be potential therapeutic strategies for Alzheimer's disease.

Investigate how the neuroinflammatory processes play a role in Alzheimer's disease pathology.